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searching for H3K9me2 16 found (29 total)

alternate case: h3K9me2

Epigenetics of cocaine addiction (3,823 words) [view diff] exact match in snippet view article find links to article

on histone modification in the nucleus accumbens. They suggest that the H3K9me2 modification in this area of the brain plays a role in the stress and depression

FOSB (7,191 words) [view diff] exact match in snippet view article find links to article

to cocaine. This depends on ΔFosB inhibiting G9a gene expression, i.e. H3K9me2 synthesis which in turn inhibits transcription factors for ΔFosB. For this

SETD6 (754 words) [view diff] exact match in snippet view article find links to article

target genes by recruiting the PKMT G9a-like protein (GLP), which catalyzes H3K9me2 and leads to chromatin silencing and gene repression. In response to stimulation

Demethylase (3,087 words) [view diff] exact match in snippet view article find links to article

KDM4A, KDM4B and KDM4C demethylate H3K9me2/3, H3K9me3 and H3K36me2/3, and KDM4D, KDM4E and KDM4F demethylate H3K9me2/3. KDM4A, KDM4B, KDM4C and KDM4D have

Epigenetics of chronic pain (2,206 words) [view diff] exact match in snippet view article find links to article

sensitivity overall. There was an increase in G9a expression and its product H3K9me2 when a nerve injury occurred. A chromatin-immunoprecipitation assay showed

Toxoplasmosis (14,650 words) [view diff] exact match in snippet view article find links to article

2010; Sun et al., 2012a). Such drug-induced downregulation of G9a and H3K9me2 also sensitizes animals to the deleterious effects of subsequent chronic

Epigenetics of schizophrenia (5,482 words) [view diff] exact match in snippet view article find links to article

prominent example is the increased repressive methylation of histone 3 (H3K9me2), which has been associated with both age of disease onset and treatment

KDM3A (229 words) [view diff] exact match in snippet view article find links to article

transcript variants. KDM3A catalyzes the demethylation of H3K9me1 and H3K9me2 residues. Its function is dependent on the presence of cofactors Fe(II)

BHLHE41 (3,158 words) [view diff] exact match in snippet view article find links to article

methyltransferase G9a. G9a then catalyzes repressive histone 3 lysine 9 dimethylation (H3K9me2) at promoter sites of target genes of MyoD. G9a also methylates MyoD, which

Addiction (25,904 words) [view diff] exact match in snippet view article find links to article

2010; Sun et al., 2012a). Such drug-induced downregulation of G9a and H3K9me2 also sensitizes animals to the deleterious effects of subsequent chronic

Treatment and management of addiction (7,006 words) [view diff] exact match in snippet view article find links to article

2010; Sun et al., 2012a). Such drug-induced downregulation of G9a and H3K9me2 also sensitizes animals to the deleterious effects of subsequent chronic

Sexual addiction (7,798 words) [view diff] exact match in snippet view article find links to article

to cocaine. This depends on ΔFosB inhibiting G9a gene expression, i.e. H3K9me2 synthesis which in turn inhibits transcription factors for ΔFosB. For this

DNA oxidation (4,790 words) [view diff] exact match in snippet view article find links to article

Abbondanza C, Avvedimento EV (January 2008). "DNA oxidation as triggered by H3K9me2 demethylation drives estrogen-induced gene expression". Science. 319 (5860):

Thomas Jenuwein (2,081 words) [view diff] exact match in snippet view article find links to article

Mechtler K, Kowalski JR, Hamon CA, Kelly TA, Jenuwein T (2007). "Reversal of H3K9me2 by a small molecule inhibitor for the G9a histone methyltransferase". Molecular

Reward system (12,762 words) [view diff] exact match in snippet view article find links to article

to cocaine. This depends on ΔFosB inhibiting G9a gene expression, i.e. H3K9me2 synthesis which in turn inhibits transcription factors for ΔFosB. For this

CCDC190 (2,085 words) [view diff] exact match in snippet view article find links to article

the microarray data on GEO database of NCBI, it can be found that the H3K9me2 methyltransferase G9a depletion can increase the expression amount of c1orf110